![]() | Only 14 pages are availabe for public view |
Abstract Variable degrees of myocardial injury can occur during the course of percutaneous coronary intervention (PCI), as reflected by rises in cardiac enzymes such as creatine kinase (CK) and CK-MB. More recently, cardiac troponins T and I have emerged as highly sensitive and specific markers of myocardial cell injury. Elevation of CK-MB after PCI is associated with increased risk of death, MI and repeat revascularization. Elevated cardiac troponin I (cTnI) after PCI is strongly associated with increased 90-day risks of major adverse cardiac events. |