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Abstract
Cirrhosis is the result of chronic liver disease that causes scarring of the liver (fibrosis and nodular regeneration) and liver dysfunction (Iredale 2003).
Sherlock and Dooley (2002) mentioned various factors contributing to the development of portal hypertension in cirrhotic patients, these include diffuse fibrosis,collagen deposition in the space of Disse,compression of intrahepatic vasculature by regenerative nodules and hepatocyte enlargement.
Portal hypertension is often obvious by clinical examination and the use of non-invasive procedures.
Determination of the aetiology and severity however requires more invasive measurements (Hoefs et al.,1994).
Duplex sonography is currently avaluable non-invasive tool in the study of cirrhosis of the liver and its complications.
Borse et al., (2002) reported that the hepatic artery resistive index is higher in cirrhotics and is related to the presence and severity of ascites.
Kok et al., (1999) stated that high resistive index of the hepatic artery is seen in patients with end stage liver disease particulary in children with cirrhosis secondry to biliary atresia..