الفهرس | Only 14 pages are availabe for public view |
Abstract Unstable angina (UA) and non-ST segment elevation myocardial infarction (NSTEMI) are two closely related forms of ACS. These two conditions share the same underlying pathophysiologic mechanism which involves the rupture (most commonly) or erosion of an atherosclerotic (atherothrombotic) plaque, with thrombus formation that severely obstructs the coronary artery lumen. Patients with these syndromes are frequently treated identically with individual variations depending on the classification of the patient as high, intermediate, or low risk. They differ primarily in-whether the ischemia is severe enough to cause sufficient myocardial damage to release detectable quantities of a marker of myocardial injury (e.g., troponin, I troponin T, or CK-MB) |