الفهرس 
ANATOMY OFOnly 14 pages are availabe for public view
 |  | from | 172 |  |  |
| | | from | 172 | | |
Abstract
INTRODUCTION
Intestinal ischaemia produces a broad spectrum of gross and
microscopic pathological changes depending on the degree and
duration of the ischaemia, its occlusive or non-occlusive nature, the
nature, the site and completeness of any occlusion, and the interval
between the ischaemic injury and the tissue examination.
In addition, secondary changes relating to bacterial invasion
vary with the portion of bowel affected and they are believed to be
more prominent in the colon.
Ischaemic lesions differ considerably tn their gross and
rntcroscopic appearance. At one extreme, there is transmural
necrosis (infarction), gnagrene, and perforation, more common tn
the small bowel than in the colon, and is associated with a
catastrophic clinical event. At the opposite extreme, and usually
more indolent clinically, are the milder lesions of mucosa] necrosis,
submucosal oedema, haemorrhage and ulceration, which ultimately
heal. Ischaemic lesions of intermediate severity can progress to
transmural necrosis or termination in transmural fibrosis and
stricture formation (Sumi Mitsudo & Lawrence J Brandt, 1992).
So, sadly, the progress for patients with some forms of
intestinal ischaemia has remained poor (Scott J Boley, Lawrence J
Brandt, /992)