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العنوان
Study of Serum levels of Zinc, Copper and Magnesium in Children with Attention- Deficit Hyperactivity Disorder /
المؤلف
Turky, Marwa Abd El-Salam Ibrahim.
هيئة الاعداد
باحث / مروه عبدالسلام ابراهيم تركي
مشرف / طارق محمد الجوهري
مناقش / مي عبدالرؤوف عيسي
مناقش / رشا محمد جمال الشافعي
الموضوع
Pediatrics. Pediatric.
تاريخ النشر
2016.
عدد الصفحات
p 163. :
اللغة
الإنجليزية
الدرجة
ماجستير
التخصص
طب الأطفال ، الفترة المحيطة بالولادة وصحة الطفل
تاريخ الإجازة
17/8/2016
مكان الإجازة
جامعة طنطا - كلية الطب - Pediatric
الفهرس
Only 14 pages are availabe for public view

from 225

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Abstract

ADHD is common long-lasting, treatable childhood neurodevelopmental
psychiatric disorder that may persist to the adulthood.
This disorder is characterized by persistent pattern of inattention and/or
hyperactivity –impulsivity that interfere with development .It has symptoms
presenting in two or more setting (e.g. at home, school, or work), and
negatively impact directly on social, academic or occupational functioning.
The symptoms must present before age of twelve and persist for more than six
months.
ADHD is considered to be a complex and heterogeneous disorder and
its etiology is not well understood, allowing the establishment of several causal
hypothesis. Evidence suggests that two main factors are associated with the
increased susceptibility of the individual to ADHD:
1. Genetic and neurobehavioral factor.
2. Environmental factor.
Some nutritional elements may be implicated in the pathogenesis of
ADHD such as Zinc (Zn), Magnesium (Mg) and Copper (Cu).
It has been suggested that symptoms of ADHD may be caused by
dopamine and serotonin dysfunction. Zinc is a cofactor for more than 300
enzymes and is involved in the pathway for the body’s production of
prostaglandins and neurotransmitters. Zinc is necessary for vitamin B6 to be
metabolized to its active form, pyridoxal phosphate, which in turn plays a role
in conversion of tryptophan to serotonin. Zinc assists in both the production
and regulation of melatonin; melatonin is an important factor in the
pathophysiology of ADHD due to its modulation of dopamine. Zinc also binds