الفهرس 
Anatomy of Rotator CuffOnly 14 pages are availabe for public view
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Abstract
Rotator cuff tendinopathy (RCT) is a leading cause of shoulder pain and
a significant source of disability and loss of work. A combination of some
extrinsic and intrinsic factors are generally responsible for the development
of RCT which begin with acute tendinitis and then progress to chronic
tendinosis and partial tears. The final stage is full-thickness tears and
complete ruptures of the tendon.
Nonoperative management is a good option of RCT without
complete ruptures while indications for operative management of RTC
disease are based largely on response to non-operative modalities and
patient goals.
Healing of repaired tendons occurred via fibrous scar tissue formation
rather than via the regeneration of a histologically normal insertion, and
thus repaired tendons have inferior mechanical properties and are more
susceptible to retear. Therefore, growth factors have been suggested to be
used to influence the healing process and promote the regeneration of the
tendon during treatment.
Platelet-Rich Plasma ”PRP” is a fraction of whole blood that contains a
concentrated amount of platelets above the baseline. Upon activation, these
platelets release a large proportion of biologically active factors that are
thought to accelerate and improve the healing process.
The total number of participants in this study was 21 patients. Eight were
males, age ranged between 24 and 62 years with the mean of 47.6. Thirteen
right shoulders were affected while 15 patients showed affection of the
dominant side. Five patients received previous corticosteroid injection, six
patients were diabetic. Regarding imaging, three patients had signs of RCT at.