الفهرس 
Preeclampsia
Differential diagnosisOnly 14 pages are availabe for public view
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Abstract
Preeclampsia is recognized as a major risk factor for cardiovascular disease later in life for both the woman and her child. Despite considerable research, the only effective treatment for preeclampsia is to deliver the baby, placenta, and all products of
conception. Maternal endothelial dysfunction is a classic hallmark of
preeclampsia. Many markers of endothelial dysfunction have been reported in preeclamptic women,including an imbalance of anticoagulation and procoagulation factors and increased levels of fibronectin,endothelial cell adhesion molecules, and other factors in the
coagulation cascade. The etiology of preeclampsia remains largely unknown. Currently, proposed causes of preeclampsia include abnormal placentation, immunologic maladaptation,inflammation,oxidative
stress,endothelial dysfunction, and genetics. The incidence of preeclampsia world wide is cited to be
5% Hypertensive disorders complicate 5 to 10 percent of all pregnancies in developed countries, 16 percent of maternal deaths
were due to hypertensive diseases. The diagnostic criteria for preeclampsia are satisfied when a woman presents with both hypertension and proteinuria, but these signs may also be accompanied by elevated serum creatinine levels, decreased platelet count of < 100,000/mm3, microangiogenic hemolysis, elevated alanine aminotransferase or aspartate aminotransferase, persistent headaches or other cerebral or visual
disturbances, or persistent epigastric pain.