الفهرس 
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Abstract
Dysphagia is described as any disruption of the normal swallow mechanism of the preparatory transport of a bolus from the oral cavity through both pharynx and esophagus to the stomach.
The pathophysiology may lie in the oral phase, pharyngeal phase, or in the esophageal phase. Several etiologic factors have been reported and categorized as morphological abnormalities, neurological disorders and general ailments. Proper history-taking and a thorough diagnostic workup using imaging studies and endoscopic examination are used to determine the exact cause.
Benign non-neoplastic lesions make up the majority of vocal fold lesions, namely Minimal Associated Pathological Lesions ”MAPLs”. They are non-inflammatory non- neoplastic lesions (polyps, nodules, cysts, Reinke’s edema, polypoid degeneration and contact granuloma).
Patients with dysphonia secondary to non-neoplastic vocal fold lesions may suffer from impaired swallowing masked by the phonatory complaint and yet is of major concern. Improved awareness regarding the prevalence of dysphagia in this group of patients may have an impact on the management strategy.
However the prevalence of dysphagia in these patients was not thoroughly investigated before.
The aim of this study is to examine the prevalence of dysphagia on basis of subjective and objective measures in patients with the presenting symptom of dysphonia and diagnosed with non-neoplastic vocal fold lesions to consider the potential benefit of swallowing therapy hand in hand with the voice therapy in the treatment of these patients if proved to have high prevalence of dysphagia.
After the application of the A-EAT-10 questionnaire on 50 dysphonic patients who were selected to participate in this study, 12 cases (about 24%) were considered dysphagic by score above 3 and underwent VFSS. Within these 12 patients, Zero percent were found complaining of dysphagia according to VFSS. So there is No Correlation between frequencies of dysphagia by A-EAT-10 and by VFSS.