الفهرس 
INTRODUCTIONOnly 14 pages are availabe for public view
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Abstract
Cardiovascular complications are already the leading cause behind the excess morbidity and mortality observed in ESRD population. Left ventricular hypertrophy (LVH) is the most frequent cardiac finding observed in this group of patients. It is mainly attributed to chronic pressure and volume overload. Renal transplantation is the best renal replacement modality offered to these patients with an expected improvement in cardiovascular related complications.
In these patients, the pathogenesis of LVH is multifactorial as several causative factors have been identified involving hypertension, hypoalbuminemia, hyperparathyroidism, anemia, chronic volume expansion and possibly hyperhomocystineamia.
Electrocardiographic study (ECG) was the first noninvasive investigation used for the diagnosis of LVH. Despite it has low sensitivity and high specificity, the worldwide accuracy of the commonly used electrocardiographic principles for excluding LVH is relatively unsatisfactory. Correspondingly, physical examination using palpation of the point of maximum impulse and assessment of the cardiothoracic index using posterior–anterior chest radiographs are quite simple, easy, and inexpensive, but unfortunately insensitive methods of assessment of LV mass.
Correction of the uremic condition by renal transplantation results in normalization of LV contractility in systolic dysfunction, reversion of hypertrophy in concentric LV hypertrophy, and enhancement of cavity volume in LV dilatation.
The aim of the study was to identify the effect of renal transplantation on the heart by studying the changes in left ventricle hypertrophy, dilatation and ejection fraction before and after kidney transplantation.