الفهرس 
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Abstract
Introduction
V
itiligo and alopecia areata (AA) are common autoimmune diseases of the skin. Vitiligo is caused by the destruction of melanocytes and results in the appearance of white patches on any part of the body. AA is characterized by patchy hair loss primarily on the scalp, but may also involve other areas as well. The two diseases appear to be quite different, targeting different cell types and managed using different treatment approaches. However, the immune cell populations and cytokines that drive each disease are similar. Vitiligo and AA have common genetic risk factors, suggesting that they share a similar pathogenesis (Harris, 2013).
Both vitiligo and AA have been described as TH1-driven diseases, based on the involvement of CD8+ T cells and the clear consistent production of IFN-γ that played a central role in pathogenesis of both diseases (Gilhar et al., 2007; Manga et al., 2016).