الفهرس 
Right coronary arteryOnly 14 pages are availabe for public view
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Abstract
Central adiposity or abdominal visceral obesity is related to metabolic syndromes such as insulin resistance, diabetes, hypertension and dyslipidaemia, which play an important role in the development of accelerated cardiovascular atherosclerosis. This association has been attributed to the enhanced lipolytic activity of visceral fat cells, with increased delivery of free fatty acids (FFAs) into the portal and systemic circulation and release of adipokines or adipocytokines related to the pathogenesis of atherosclerosis Epicardial fat (EF) is the adipose tissue around the heart constrained by the visceral pericardium and has the same origin as abdominal visceral fat. It encases the coronary arteries without an intervening fascial barrier and also shares common innervation and blood supply with the coronary arterial wall. Due to this unique anatomic relationship, it have been suggested that EF is strongly related to the development of coronary artery disease (CAD) 4,5 The accumulation of EF is known to be a rich source of free fatty acids and a number of inflammatory Cytokines and pro-atherogenic mediators that aggravate vessel wall inflammation and stimulate the progression of atherosclerosis from (outside to inside) and thus directly influence the development and progression of CAD through a local paracrine effect 6,7. Previously, the association between epicardial fat thickness and CAD has been.