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Abstract
Introduction: Systemic lupus erythematosus (SLE) is an autoimmune disorder which has multi-organ involvement. The continuation of inflammation in lupus could be attributed to failure of the resolution process due to deficiency of potent endogenous pro-resolution-inducing molecules such as lipoxin A4 (LXA4) leading to progression and flares of lupus and lupus nephritis. Objective: The aim of this study is to assess the levels of serum and urinary LXA4 in SLE patients and in healthy controls, and to correlate them with various clinical and laboratory data as well as renal biopsy and disease activity indices