الفهرس 
IntroductionOnly 14 pages are availabe for public view
 |  | from | 260 |  |  |
| | | from | 260 | | |
Abstract
The mechanism by which varicocele alters the testicular physiology has been very elusive. Over the years, as number of theories have been put forth in an attempt to elucidate the causes of varicocele¬induced impaired spermatogenesis. However, none of these theories have been proven to be operative. Likewise, the controversial data available today do not allow us to explain why testicular function improves after varicocelectomy. This randomized prospective study was carried out on 100 varicocele patients admitted to Endocrine Surgery Unit at Mansoura University Hospital during the period from October 1995 to June 1997. All varicocele patients, both married (fertile and infertile) and unmarried, were having varicocele-induced impaired spermatogenesis that was reflected on conventional semen analysis by one or more criterion of the stress pattern triad (oligozoospermia, asthenozoospermia, and teratozoospermia) . Patients fulfilling the selection criteria were randomly allocated to three groups and subjected to inguinal varicocelectomy (group I; 40 patients), high ligation varicocelectomy (group II; 40 patients) and laparoscopic varicocelectomy (group III; 20 patients). Postoperative follow up was continued for two years for all patients and the rate