Search In this Thesis
   Search In this Thesis  
العنوان
New approaches for controlling the cotton leafworm spodoptera littoralis Boisd /
المؤلف
Mohamed, Maaly Hassan.
هيئة الاعداد
باحث / معالي حسن محمد
مشرف / سعيد أحمد عمارة
مشرف / جمال عبدالحميد
مشرف / محمد محمود البكل
الموضوع
the cotton leafworm. Boisd.
تاريخ النشر
2004.
عدد الصفحات
86 p. :
اللغة
الإنجليزية
الدرجة
ماجستير
التخصص
علم الحيوان والطب البيطري
تاريخ الإجازة
1/1/2004
مكان الإجازة
جامعة المنصورة - كلية العلوم - قسم علم الحيوان
الفهرس
Only 14 pages are availabe for public view

from 105

from 105

Abstract

The insecticidal, biological and morphogenetic effects of flufenoxuron Bacillus thuringiensis (Agrien) and cyanophos, as well as the effect of the two most active compounds ( flufenoxuron, Bacillus Thuringiensis) on the histology of the mid gut and integument of 6th instar larvae of Spodoptera littoralis treated as 4th instar larvae with LC50 of the previous compound. The insect growth inhibitor flufenoxuron was the most toxic compound against the larvae of 2nd and 4th instar larvae of S. littorali. Both compounds of flufenoxuron and Bacillus thuringiensis tested significantly increased (P< 0.05) the mean larval and pupal durations. On the other hand both compounds decreased the percentage of pupation, adult emergence, the mean pupal weight, adult longevity, fecundity and fertility. However the pre­oviposition, oviposition and post­oviposition periods differed according to the compound used. Both compounds of flufenoxuron, and Bacillus thuringiensis (Agerin) exhibited larval, pupal and adult deformities as well as pupal­adult intermediates. Treatment of 4th instar larvae of S. littoralis with flufenoxuron and Bacillus thuringiensis resulted in sever histological changes in the integument and midgut of surviving late 6th instar larvae. Flufenoxuron disrupted formation of endocuticle whereas Bacillus thuringiensis (Agerin) effected only the hypodermis. The most prominent changes induced by these two compound for the midgut was the vacuolation of epithelial cells and disruption of peritrophic membrane.