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Abstract Cardiac troponin-I (Tn-I) is a sensitive and specific marker for detecting myocardial damage. Elevation of cardiac troponin was detected in 30% to 40% of patients undergoing catheter based coronary interventions. This rate may be higher than CK-MB elevations which has been reported in 5% to 26% of patients undergoing percutaneous transluminal coronary angioplasty. Myocardial necrosis after coronary angioplasty has been traditionally based on CK-MB level (Shmuel et al., 2000). Previous studies suggested that CK-MB elevation after coronary angioplasty is associated with increased late mortality. However other studies found no adverse long term clinical outcomes in patients with CK-MB elevations post coronary angioplasty (Baims et al., 1998). It was found that conjoint elevation of Tn-I and CK-MB levels after coronary angioplasty are associated with increased risk of major in-hospital complications but have no incremental risk of intermediate-term clinical outcomes (Shmuel et al., 2000). |