الفهرس 
Material and methodsOnly 14 pages are availabe for public view
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Abstract
Carbon monoxide poisoning is one of the leading causes of
morbidity and mortality from poisoning involves myocardial and/or
neurological injury.
This work was conducted on 40 (24 males and 16 females) CO
poisoned patients and 10 normal persons were chosen as a control group.
They were chosen randomly among those attending to the Poison Control
Center, Ain Shams University.
This work aimed at correlating the clinical state with COHb%
levels. All patients as well as the control were subjected to detailed history
taking, clinical examination, blood sampling for blood COHb%, serum
AST, LDH, CPK-:MBconcomitant with electrocardiogram. 22 cases were
categorized as patients with ECG changes necessitating the admissionin
intensive care unit. 18 cases categorized as patients without ECG changes
admitted as inpatients.
The vomiting and headache were the commonest symptoms of CO
poisoned patients. Other findings as coma as a sign of severity and
tachycardia were seen. There was a significant DROP in B.P. in the
intoxicated cases with CO exposure. ECG changes were in the form of ST
segment depression or elevation and T wave flattening or inversion.No
correlationbetween all these clinical data and COHb%.
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Summary, Conclusion anti ~mentfations
The cardiac enzymes CPK~MB and AST were significantly elevated
in patients with ECG changes indicated myocardial ischemic insult.
Recommendations:
1. COHb% is the important test -to diagnose CO poisoning but no
correlation with severity of cases or the treatment strategy.
2. ECG and cardiac enzymes are mandatory in CO poisoned patients
especially comatosed patients to diagnose myocardial ischemic insult.